Thanks again for discussing this with me. I’d like to confirm your previous comment:

(1) “Look at mitochondrial DNA cleavage codons in zoological jump species to humans and the same in S and L type in humans. Why this novel virus could have “ mutated” in Asian civet cats from SARS bat independent.”

(2) “More like vectoring into virus from DNA fragments that occur during cell mitosis with mitochondrial DNA.”

Do you mean that during cell division in civets, the civets’ mitochondrial DNA might’ve been integrated into SARS, giving rise to SARS-CoV-2? Or this could occur with pangolins’ mitochondrial DNA?

I think it’s a real mystery how SARS-CoV-2 got into humans. A 14th May published paper suggests that pangolins might not be the intermediate host after all. Maybe there are really two intermediate hosts…