For the second question, I came across this preprint that found that:

"Both smoking and use of nicotine and flavor-containing e-cig led to upregulations of pro-inflammatory cytokine production and expression of genes related to inflammasomes."

Link: https://www.biorxiv.org/content/10.1101/2020.07.13.198630v1

So, it seems that as long as the product contains nicotine, it can increase the expression of ACE2 receptor.

But there are some studies that found that smoking or COPD (chronic obstructive pulmonary disease; almost always caused by smoking) decreased ACE2 receptor expression instead. So, I think the effects of smoking/nicotine/COPD on ACE2 are still inconclusive. You read more about this here: https://www.sciencedirect.com/science/article/pii/S030698772100147X